HBOT and Inflammation Response

HBOT saturates the plasma with oxygen, resulting in increased oxygen delivery to the tissues.  HBOT dissolves oxygen into the plasma, increases oxygen tension in hypoxic areas, enhances white blood cell activity at the wound site, reduces edema by vasoconstriction, blocks cytotoxic effects of carbon monoxide and hypoxia associated with cyanide poisoning and starts the formation of new peripheral vascular vessels and nerve endings (angiogenesis) in damaged areas of the body.

According to Harch, “high pressure oxygen delivered within two to three hours after the injury, significantly quenches the secondary destructive inflammatory reaction… and major damage is minimized.”21 Since 1993 research has provided the evidence to explain the oxygen paradox:  HBOT quenches the inflammatory reaction.

Notes
21Harch, 26.